冠状动脉疾病
端粒
心脏病学
内科学
计算机辅助设计
医学
人口
线粒体DNA
生物
遗传学
DNA
基因
生物化学
环境卫生
作者
Georgios Rampidis,Georgios Benetos,Dominik C. Benz,Andreas A. Giannopoulos,Ronny R. Buechel
标识
DOI:10.1016/j.atherosclerosis.2019.05.012
摘要
We have read with great interest the recent article by Wang et al. [1], in which the researchers investigate the effects of leukocyte telomere length and mitochondrial DNA copy number, alone or in combination, on coronary artery disease (CAD) risk and severity in the Chinese population. As reported by the authors, the angiographic burden of CAD was quantified by the modified Gensini Score.
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